DSA
Anabolikler & SARMİyi Çalışılmış5 çalışma

Oxandrolone

Oral Anabolic Steroid | Lean Mass & Recovery

Oxandrolone is a synthetic oral anabolic-androgenic steroid derived from dihydrotestosterone (DHT), first synthesized in 1962 by Raphael Pappo at Searle Laboratories and introduced to the market in 1964 under the brand name Anavar. It was designed to be a mild anabolic agent with minimal androgenic activity, achieved through a structural modification where an oxygen atom replaces the carbon-2 atom in the A-ring of the DHT backbone. This modification significantly reduces androgenic potency while preserving anabolic effects on skeletal muscle. Oxandrolone is FDA-approved for the promotion of weight regain following involuntary weight loss due to surgery, chronic infection, severe trauma, and prolonged corticosteroid use, and for the relief of bone pain associated with osteoporosis. It has been extensively studied in burn recovery, HIV/AIDS-related wasting, and pediatric growth disorders. Among oral anabolic steroids, oxandrolone is considered one of the mildest with respect to hepatotoxicity and virilizing side effects, which has made it one of the few anabolic steroids used in women and children in clinical settings.

Yarı ömür
~9-10 hours
Sıklık
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Döngü
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Doz
20-50 mg/day (male), 5-20 mg/day (female)
Oxandrolone 2D chemical structure

M.W.: 306.44 Da

PubChem 2D yapı diyagramı

DEEP ARCHIVESUPPLEMENTOxandroloneOral Anabolic Steroid | Le…PURITY: 99% HPLCRUO: Research Use OnlyREF OXA-361-DSAM.W. 306.44 DaNOT FOR HUMAN OR VETERINARY USE

Referans illüstrasyonu — ürün listesi değildir

Etki Mekanizması

Oxandrolone exerts its anabolic effects primarily through binding to the intracellular androgen receptor (AR), promoting nitrogen retention and protein synthesis in skeletal muscle tissue. As a DHT derivative, it cannot be converted to estrogen by the aromatase enzyme, which means it does not cause estrogen-mediated water retention or gynecomastia. The 2-oxo modification in the A-ring reduces its affinity for 5-alpha reductase, limiting conversion to more potent androgens in peripheral tissues and contributing to its relatively low androgenic profile. Oxandrolone increases protein synthesis through direct AR-mediated gene transcription, enhances the expression of genes involved in muscle hypertrophy and repair, and promotes collagen synthesis. It has also been shown to reduce cortisol-binding globulin levels and attenuate the catabolic effects of glucocorticoids, which is particularly relevant in burn patients and those recovering from severe trauma. Additionally, oxandrolone stimulates hepatic production of sex hormone-binding globulin (SHBG) to a lesser degree than other oral steroids, though it does suppress endogenous testosterone production through negative feedback on the hypothalamic-pituitary-gonadal axis.

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Oxandrolone is a synthetic oral anabolic-androgenic steroid derived from dihydrotestosterone (DHT), first synthesized in 1962 by Raphael Pappo at Searle Laboratories and introduced to the market in 1964 under the brand name Anavar. It was designed to be a mild anabolic agent with minimal androgenic activity, achieved through a structural modification where an oxygen atom replaces the carbon-2 atom in the A-ring of the DHT backbone. This modification significantly reduces androgenic potency while preserving anabolic effects on skeletal muscle. Oxandrolone is FDA-approved for the promotion of weight regain following involuntary weight loss due to surgery, chronic infection, severe trauma, and prolonged corticosteroid use, and for the relief of bone pain associated with osteoporosis. It has been extensively studied in burn recovery, HIV/AIDS-related wasting, and pediatric growth disorders. Among oral anabolic steroids, oxandrolone is considered one of the mildest with respect to hepatotoxicity and virilizing side effects, which has made it one of the few anabolic steroids used in women and children in clinical settings.

Moleküler Veri

Tür
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Moleküler ağırlık
306.44 Da

1000 mcg = 1 mg

1 = her gün · 7 = haftalık

Süre (gün)

Araştırma Endikasyonları

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most effective

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most effective

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effective

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effective

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effective

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effective

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effective

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effective

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effective

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moderate

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Doz Protokolleri

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HedefDozSıklıkYol
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Etkileşimler

++
Testosterone
synergistic

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+
Finasteride
compatible

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+
N-Acetylcysteine (NAC)
compatible

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+
TUDCA
compatible

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~
Warfarin
monitor

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~
Oral Hypoglycemics
monitor

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++
Growth Hormone
synergistic

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~
Corticosteroids
monitor

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Ne Beklenmeli

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Yan Etkiler & Güvenlik

Yaygın Yan Etkiler

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Durdurma Belirtileri

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Kontrendikasyonlar

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Sık Sorulan Sorular

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